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Eat Your Greens, or Your Gut Gets It 

While Congress lets Big Food crush school lunch reform, new research shows just how bad the status quo is. By Tom Philpott | Wed Nov. 16, 2011 4:00 AM GMT While Big Food rams its Tater Tots and frozen pizza school lunch agenda through Congress [1], we're learning more about the effects of diets high in starchy foods and low in green vegetables. And it's not pretty. I pointed yesterday to a vast recent Harvard study [2]finding that heavy consumption of potatoes—even in nonfried forms—leads to unhealthy weight gain. Now, from UK scientists, comes a study (press release here [3]; abstract here [4]) suggesting that green vegetables may have even more dietary importance than we previously thought. (Hat tip Atlantic Life [5].) 

The researchers subjected mice to a diet stripped of vegetables and found that after just three weeks, the mice lost 70 to 80 percent of a kind of white blood cell called intraepithelial lymphocytes, which, the press release states, "play a critical role in monitoring the large number of micro-organisms present in the intestine, keeping infections at bay and maintaining a healthy gut." Also read: How the beef and dairy industries make sure school lunches stay loaded with fat. [6] The researchers posit that a substance known as indole-3-carbinol, prominent in leafy greens, is responsible for maintaining these white blood cells. Take it out of the diet, apparently, and the cells die. Here's a graphical depiction of their findings: One of the researchers, Marc Veldhoen, remarked that, "since the new diet contained all other known essential ingredients such as minerals and vitamins," the results surprised him. But I'm not surprised at all. 

Foodstuffs are complex; they are not the sum of their vitamins and minerals, calories and fiber, fat and protein, or any other isolated substance currently being fetishized or demonized by the food industry. As this study shows, you can't calculate the level of vitamins and minerals found in leafy greens, synthesize them, combine them in a vitamin pill, and then happily dispense with leafy greens. Whole foods interact with our bodies in ways we are only beginning to understand. I predict someone will be inspired by this study to isolate indole-3-carbinol, synthesize it for a mass-produced pill, and market it as an immune-enhancing wonder supplement. If it happens, I'm willing to bet that that researchers will find that indole-3-carbinol supplements don't do the work of leafy greens, either. Recall that when scientists discovered the benefits of antioxidants found in fruits and vegetables, the supplement industry rushed out with all manner of antioxidant potions—which proved to be worthless [7]. It turns out that isolated beta-carotene added to a pill or a can of soda doesn't offer the same benefits as beta-carotene in the context of a carrot. Unfortunately, a recent Centers for Disease Control and Prevention study [8] found that more than two-thirds of US adults fail to meet the recommended daily intake of at least five fruits and vegetables per day. "Eat real veggies" is something we could be teaching kids in school cafeterias. Instead, we're going to keep teaching them to scarf down stuff like "potato smiles [9]." 

Source URL: http://www.motherjones.com/tom-philpott/2011/11/eat-your-greens-or-your-gut-gets-it Links: [1] http://motherjones.com/tom-philpott/2011/11/school-lunch-frozen-pizza-tomato-paste [2] http://www.nejm.org/doi/full/10.1056/NEJMoa1014296 [3] http://www.babraham.ac.uk/news2011/oct-13.html [4] http://www.cell.com/abstract/S0092-8674%2811%2901136-6 [5] http://www.theatlantic.com/life/archive/2011/11/eat-your-green-vegetables-how-to-keep-your-intestinal-tract-healthy/248250/ [6] http://motherjones.com/politics/2003/01/unhappy-meals [7] http://www.ncbi.nlm.nih.gov/pubmed/15153272 [8] http://www.cdc.gov/mmwr/preview/mmwrhtml/mm5935a1.htm [9] http://www.schoollunchsolutions.com/documents/3456PotatoSmiles.pdf
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Aluminum & Dementia, fluoride reaction emphasized

Doug Cross is a chartered biologist, who fought for victims of the waterpoisoning incident when 20 tonnes of aluminium sulphate were put in the wrong tank in Camelford, Cornwall, in 1988. He is a BSc (Hons) PGCE, CBiol MiBiol, EurBiol. 
He writes: below is the text of my web page dated 20th May 2012,summarising recent developments in our understanding of the link betweenaluminium and Alzheimer' s Disease, and the very real concern of some of our most eminent research specialists over the potentially lethal obsession of the dental public health sector of making the situation even worse by adding fluoride to aluminium-contamina ted water! The title refers to two of the greatest threats to public health - I have not included the second (the impact of using alumninium hydroxide as an adjuvant in vaccines) as it's not relevent to the fluoridation debate - but it's worth being aware of this as well! Pass this on if you wish - the vaccine article can be found on the same web page indicated below. Doug.

http://www.ukcaf. org/a_lethal_ addiction_ -_aluminium_ alzheimers_ and_aut.html

The new paradigm - aluminium may be responsible for two of the greatest threats to public health.
Doug Cross
20th May 2012 

The most persuasive argument against water fluoridation is that fluoride can contributes to the epidemic of dementia. Whilst proponents and opponents of fluoridation argue over the minute details of its direct impacts on health, an almost unnoticed revolution in the understanding of dementia in the elderly is set to render such debates trivial in comparison. New research into the relationship between aluminium poisoning and dementia has suddenly thrown a new spotlight on the contribution that water fluoridation could be making to the future burden of dementia within our society. We ignore this new understanding at the peril of both ourselves and our descendants.

The prevalence of dementia, and particularly Alzheimer' s Disease (AD), is already a far greater health concern than cancer, and it's expected to triple in the next thirty years. For years the cause of AD has been controversial, and the main contender as the causative agent, aluminium, has been a matter of hot dispute. "It' s the third most common element in the Earth's crust," these environmentally illiterate ignoramuses claim, "It can't possibly be toxic!" Well it is, and now we have the proof. Now things are about to change. In an authoritative review published last year in the highly regarded Journal of Alzheimer' s Disease, Lucija Tomljenovic concludes unequivocally that aluminium accumulation in brain tissue over a lifetime is a leading cause of the onset of Alzheimer' s Disease in the elderly.

She warns that "In stark contradiction with the abundance of research evidence . . there appear to be "several hostile intellectual attitudes" that reject the possibility that Al toxicity contributes to the growing incidence of AD. She says that the evidence that aluminium is not merely associated with AD, but actually contributes significantly to its development, "is built upon very solid experimental evidence and should not be dismissed." 
 
Most of the bioavailable aluminium that accumulates in our brains comes from our drinking water. That's right - it appears that the almost universal use of aluminium sulphate in modern water treatment works may actually constitute one of the greatest health threats to the older people in our

As Tomljenovic says, resistance to the evidence that aluminium is a causative agent for dementia is extremely dangerous: "Such widely circulated opinions hamper implementation of preventive plans to lessen exposure to aluminium which, according to some leading scientists advice, would be the most sound and cost-effective approach to the growing incidence of Alzheimer's dementia." 

Why is this of interest to the anti-fluoridation community - and of course, those intellectually paralysed health professionals and politicians who still think that it's a great policy? Because anything that increases theability of aluminium to get into our bodies is liable also to be capable of accelerating the onset of dementia in old age - and fluoride is the most effective substance that increases its absorption from the environment.

Tomljenovic describes the problem very clearly: "In the presence of fluoride, only trace amounts of Al are needed to produce substantial neuronal injury. Both fluoride and Al when complexed in AlF-x [fluoroaluminates] appear to be more easily absorbed from the GI [gastrointestinal] tract compared to their ionic forms."

She is particularly concerned over the practice of water fluoridation: "In spite of these observations, water fluoridation persists in USA, Canada, Australia, and New Zealand, whilst most of Europe has abandoned this practice. . . .In fluoride-treated water, fluoroaluminates are the prevalentspecies. The enhanced transport of fluoroaluminates across the GI tract and the BBB [blood-brain barrier], in context to their highly neurotoxic potential, raises significant concerns about the prevalence of these compounds in drinking water of various countries" .

The practice of adding to our drinking water the very substance that is most likely to increase the risk of aluminium intoxication leading to Alzheimer' s Disease in the elderly - fluoride - could not represent a more direct threat to the future welfare of increasing numbers of our elderly. Already one in five over the age of 80 develops dementia, and things are only going to get worse.

It is essential that we get rid of the aluminium in our public drinkingwater supplies, and stop stupidly adding fluoride to it. The evidence that the cumulative effects of chronic aluminium intoxication are responsible for this and related disastrous medical condition is now overwhelming, and causation established beyond reasonable doubt. Claiming that there remains any uncertainty about aluminium as a primary cause of AD, in the face of the convincing evidence that it is, is as perverse as the persistent andperverse fiction that fluoride is 'safe and effective' as a prophylactic treatment for dental decay.

Now it is apparent that two rampant forms of man-madeenvironmentally- mediated epidemic disease - dental fluorosis and dementia - could quite easily be brought under control. But this requires both the health and water sectors to recognise their responsibilities and take appropriate action.

Dental fluorosis could be brought under control within a decade or so, as the last vulnerable children pass through adolescence and experience the degradation of their teeth caused by their unfortunate exposure to fluoride during their earlier childhood.

But even if we take action now to replace the aluminium-based flocculants that are almost universally used in modern water treatment with less hazardous substances, most of us will not see a significant reduction in the prevalence of Alzheimer' s Disease within our own lifetimes. The latent development period for dementia appears to be sixty to seventy years, suggesting that exposure over many decades eventually causes an inexorable progression towards the onset of this dreadful result of chronic poisoning. 

But that is no excuse for failing to act now - this is truly a just case for the imposition of the Precautionary Principle, for the future benefit and protection of those who will come after us. 

Lucija Tomljenovic. Aluminum and Alzheimer' s Disease: After a Century of Controversy, is there a Plausible Link? Journal of Alzheimer' s Disease 23(2011)567- 598. (Abstract from http://www.ncbi. nlm.nih.gov/ pubmed/21157018 ,0,'yes&# 39;,' yes') ;> )

DISCLAIMER: The issues raised by these papers are highly controversial within certain sectors of the medical and pharmacological sectors. The comments in this summary reflect my own personal interpretation of the reference sources identified here. They do not purport to provide medical opinion, and should not be taken as such. Anyone who may be concerned over their own or their family's medical welfare should consult a qualified medical practitioner - please do not contact me, as I am unqualified tooffer any advice in medical matters.
Doug Cross
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